Cetyl Mobility and Other
Products for Auto Immune System, Arthritis, Fibromyalgia Disorders
PDF Fact Sheet on Cetylmobility
This natural synergistic nutrient formula of essential
minerals, long-chain fatty acid methyl esters, and Type II collagen enhances
mobility for the moving joints of the body, restoring free movement and
providing structural material in rebuilding healthy joint cartilage. The result
of increased joint lubrication and cartilage reformation is to reduce the
nagging effects of overused, aged or strained joints. The fatty acid cetyl ester
formulation seems to function first as an effective lubricant for the joints.
The Type II collagen in this nutrient formula supports the careful
reconstruction of damaged joint tissues, including tendons, ligaments, and
Magnesium Chelate: Important mineral in binding ATP. Required
for phosphorylation reactions in glycolysis. The proper activity of numerous
Mg-requiring enzymes is effected by the magnesium level in blood. Magnesium is
required at various steps in the synthesis of DNA, RNA, and protein.
Zinc: Biological catalyst in many enzyme systems.
Speculation: zinc supplements raise the zinc level within the joints themselves,
thus having an anti-inflammatory effect at the very site of trouble.
Copper: This mineral reduces swelling and redness on inflamed
joints. Copper activates superoxide dismutase, a powerful free-radical
Manganese: National Research Council set adequate dietary
intake at 2.5-5 mg/day. Mn necessary for enzymatic syntheses of
mucopolysaccharide, which is deficient in rheumatoid arthritis. Mn is a trace
mineral, plays role in activating numerous enzymes. It acts as a catalyst in
synthesizing fatty acids. It is necessary for normal skeletal development.
Cetyl Myristoleate (C16 ester of cis-9-unsaturated C14 fatty
acid): Cetyl myristoleate acts as a joint lubricant. It enhances joint mobility
by thinning the natural joint lubricants that may have hardened due to aging and
stress. Enhances mobility while a healthy balance of
collagen/mucopolysaccharide/water in joint cartilage is being achieved through
proper nutrition and dietary supplementation.
Cetyl myristoleate acts first, as a surfactant to lubricate
synovial fluids, second, to regulate prostaglandin and leukotriene production to
decrease inflammation, and last, as an immuno-modulator of hyper-immune
responses such as that found in rheumatoid arthritis.
In a multicentered double-blind, randomized,
placebo-controlled parallel trail of 431 patients
is reported: “The results of this trial suggest that cetyl myristoleate and
cetyl myristoleate supporting formulas may be beneficial in the treatment of
many forms of arthritic based diseases, including psoriatic arthritis.” “Cetyl
myristoleate & supporting formulas produced the best treatment response by a
factor of 72.8% more patients than did placebo.” “...it is obvious that there is
a statistically significant improvement in the use of the cetyl myristoleate
with supporting formulas.” This trial “...showed the majority of patients
responding to cetyl myristoleate and cetyl myristoleate supporting formulas did
so within the first three weeks.” “Cetyl myristoleate and cetyl myristoleate
supporting formulas were well tolerated in this trial. This finding is not
unexpected as cetyl myristoleate and the cetyl myristoleate supporting formula
components are naturally occurring and have been used as diet supplementation
for many years and are widely available singularly and in various combinations.”
“In summary, cetyl myristoleate and cetyl myristoleate supporting formulas
appear to be beneficial in the treatment of a wide range of arthritic conditions
including long standing and refractive cases.” (Siemandi 1997)
Cetyl Myristate (C16 ester of C14 fatty acid): Cetyl
myristate acts as a joint lubricant.
Cetyl Palmitoleate (C16 ester of cis-9-unsaturated C16 fatty
acid): Cetyl palmitoleate acts as a joint lubricant.
Cetyl Laurate (C16 ester of C12 fatty acid): Cetyl laurate
acts as a joint lubricant.
Cetyl Palmitate (C16 ester of C16 fatty acid): Cetyl
Palmitate acts as a joint lubricant.
Cetyl Oleate (C16 ester of cis-9-unsaturated C18 fatty acid):
Cetyl oleate acts as a joint lubricant.
Type II Collagen: Contains high concentrations of
mucopolysaccharides: glucosamine sulfate and chondroitin sulfate. Protects
cartilage as well as rebuilds and stimulates the production of cartilage. Type
II collagen is derived from the soft sternum cartilage of young chickens.
Collagen II supplies enzyme inhibitors, including proteoglycans, glucosamine,
and chondrointin, which stop cartilage loss caused by enzymatic degradation.
Proteoglycans are hygroscopic – assisting to attract moisture to repair joint
cartilage. Other components of collagen II are anti-angiogenic to minimize joint
redness. Research has also isolated growth factors in collagen II that help
regeneration of joint cartilage.
Collagen II mucopolysaccharides in cartilage ease swelling
and improve and accelerate wound healing. The mucopolysaccharide component of
cartilage is the site of initial destruction in many joint disorders. Collagen
II is a powerful alleviator of joint discomfort and a potent regenerator of
cartilage tissue. Collagen II enables the body to build strong, new structural
support. Type II collagen knits bone and provides elasticity to joints, serving
as a shock absorber.
Clinical trial of oral administration of Type II collagen to
60 patients with randomized, double-blind study
reported: “This controlled trial provides evidence that oral administration of
small quantities of solubilized native heterologous type II collagen is both
safe and can improve the clinical manifestations of active rheumatoid
arthritis.” “...this study demonstrates the therapeutic efficacy of oral
tolerance for a human autoimmune disease and provides the foundation for the
development of oral collagen as an easily administered nontoxic treatment for
rheumatoid arthritis.” “If longer term efficacy is established, oral collagen
would be a preferable treatment because it is not toxic.” “There was no evidence
of sensitization to collagen, as measured by antibodies to type II collagen.”
Clinical trial (multi-site, double-blind, placebo-controlled
with 274 patients) of oral administration of Type II collagen reported:
“Overall, there were no differences between the treatment groups in the
incidence of any adverse event during this study, and no serious adverse event
was attributed to CII (Type II collagen).” “There was no significant change in
IgA or IgG antibody status with treatment, suggesting that sensitization to CII
(Type II collagen) did not occur.” “When the aggregate group of all patients
treated with any dosage of CII (Type II collagen) was compared with the total
placebo group, the presence of anticollagen antibodies was significantly
associated with an increased likelihood of achieving a clinical response to CII
(Type II collagen).” “The phenomenon of oral tolerance represents a novel
approach to the treatment of chronic, disabling autoimmune diseases. To date, no
serious adverse events have been noted in animal or human studies of oral
tolerance (of Type II collagen), and the simplicity and apparent safety of this
form of treatment make it extremely appealing.”
Blind cetylmobility Testing by Dr. Jorge Flechas
Cetyl Test Page 1
Cetyl Test Page 2
Cetyl Test Page 3
Cetyl Test Page 4
Omega-3 Fatty Acids (Omega-3 EPA)
Have strong anti-inflammatory properties.
"Also, researchers at Harvard University found that 1.8 grams
of EPA greatly improved tenderness in the joints, and morning stiffness in
individuals suffering from rheumatoid arthritis (Quillin 1987). Improvements in
this study group occurred most noticeably within 12 weeks. The results of this
study can be attributed to the fact that when EPA and DHA are high in the diet,
there is a corresponding reduction in leudotriene production. As previously
noted, leukotrienes cause inflammation in the tissues."
MSM (methylsulfonylmethane) (Anatomix)
This rich source of natural sulfur is an integral component
of healthy joint cartilage. MSM is a naturally occurring organic source of
nutritional sulfur and is found in all living organisms. Although MSM is found
in many fresh fruits, vegetables, milk and grains, it is purportedly destroyed
by food processing, cooking and storage. MSM is utilized daily by our bodies.
Good health practices involve replacing essential substances that our bodies
naturally deplete or diminish through illness or abuse. Maintaining elasticity
and flexibility of joint cartilage requires adequate levels of MSM. In addition,
MSM has been linked with healthy nutrient transport across the cell
protein/lipid membrane. This enhanced flow eliminates harmful products from
cells, while permitting uptake of key nutrients to promote healing. By reducing
the pressure in cells, redness and swelling of joint tissues is reduced.
Vitamin B-1 (in
"This vitamin helps prevent crosslinking - the abnormal
bonding and stiffening of collagen and elastin, the ‘protein glue’ in the white
fibers of our tendons, bones, cartilage, skin, and all other connective tissue"
Vitamin B-3 (niacin, nicotinic acid, niacinamide, inositol
hexanicotinate) (in Maximol)
Is a vasodialator (assists in widening arteries) to promote
blood flow. "Early research by Dr. William Kaufman (1949) had demonstrated
niacin's ability to diminish the pain associated with arthritis" Niacin is
intimately involved with more than 50 metabolic reactions in the body.
Vitamin B-5 (calcium pantothenate) (in Maximol)
Involved in Krebs Cycle.In a study by Dr. David W. Twickenham
(1980, p211) "…highly significant effects were recorded for calcium pantothenate
(B-5) in reducing the duration of morning stiffness, degreee of disability and
severity of pain." The subjects in the control group who received a placebo did
not experience any such effects.
Vitamin B-6 (in
According to Kronhausen and associates (1989), this nutrient
helps with arthritic discomfort by shrinking swollen joint membranes and
dissolving rancid fat deposits. Pain is reduced and the sufferer regains some of
the lost mobility in the joints.
Selenium (in Maximol)
Selenium is a component of the protective enzyme glutathione
peroxidase, a powerful free-radical scavenger."…while in rheumatoid arthritis,
superoxide radicals and lipoperoxides which can be generated in the tissues and
accelerate the progression of the disease,…selenium can slow down this process."
(Passwater, 1980, p85-87)
Indications and Usage:
Take two capsules twice daily morning and evening for 60
days. Reduce to one capsule once or twice daily for maintenance.
For children, start with half the daily recommended dosage
and slowly increase to the regular dose.
Contraindications and Warnings:
Some patients (less than 1%) receiving cetyl myristoleate
treatment experience mild stomach discomfort, nausea or loose stools. This is
usually caused by the inability to digest fatty acid ester oils. This can
usually be remedied by supplementing the diet with lecithin to emulsify the oil,
and digestive enzymes, particularly lipase, which helps break down the fatty
acid ester oils.
May not be very effective if there is a known liver
dysfunction. Cholecystoctomy patients or those with a history of liver or gall
bladder disease or obstruction may also have trouble absorbing cetyl
myristoleate. These patients will require lecithin and lipase to emulsify the
May take as long as 10-14 days for signs of improved free
mobility, longer in some cases. During the supplement period of 3-30 days, most
patients experienced enhanced joint mobility and reduction in joint pain. Many
benefited from continual small daily maintenance doses without re-experiencing
joint mobility problems and pain.
Recommended diet: Eat more uncooked fruits and vegetables for
vitamin, minerals and antioxidants. Eat more cold water fish (salmon, herring,
mackerel, albacore tuna) as a consistent source of omega-3 fatty acids. Eat less
cooked animal meat to minimize consumption of arachadonic acid, which increases
joint inflammation. Drink at least 2-3 quarts of water every day to hydrate the
body and keep the cells functioning properly.
Mobility the Product
PDF Fact Sheet on Cetylmobility
 H. Siemandi, et al, (1997). “The effect of cis-9-cetyl
myristoleate (CMO) and ajunctive therapy on the course of arthritic episodes in
patients with various auto-immune diseases characterized by the common
terminology, ‘arthritis’ and ‘psoriasis’”,
Townsend Letter for Doctors & Patients Aug./Sep. 1997 #169 p. 58-63.
 D. Trentham, et al, (1993). “Effects of oral
administration of type II collagen on rheumatoid arthritis.” Science, 261 (5129) 1727-30.